Position Summary:
Researcher will have experience in the HIV field to study the mechanisms of HIV persistence and latency in HIV infected subject treated with highly active antiretroviral therapy (HAART). The program includes in vitro and ex vivo studies on human samples with particular focus on latently infected cells.  The overarching goal of this project is to develop novel therapeutic strategies aimed at eradicating HIV.

Duties:

• To develop and validate primary cell models of HIV latency aimed at identifying molecules endowed with the ability to induce viral production from cellular reservoirs
• To identify the cellular pathways involved in HIV latency using siRNA technology in human primary CD4 T cells 
• To identify virological and immunological biomarkers of HIV persistence in human samples including lymphoid tissues 
• Supervise a research assistant who will contribute to the development of the project
• Work in close collaboration with other scientists involved in related projects
• Analyze and integrate data, present data in conferences, write papers
• Other project related duties to sustain its efficiency 

Minimum Qualifications:

• 3 years experience in the study of immunology and/or HIV
• Experience in the HIV latency/reservoir field is a plus
• Ability to work as a team player in highly interactive and multidisciplinary research environment

• Ability to record and communicate experimental details under minimal supervision and to produce quarterly reports
• Responsible, hardworking individual who’s able to multitask and to work independently 
• The candidate should have good problem solving skills, be innovative and highly organized 
• Excellent communication skills in English language

Education:

• PhD in immunology or microbiology
• 3 years lab experience in the techniques of primary cell culture in BSLII + environment, cell signaling (western blot), virological assays (PCR, RT-PCR)

Please apply by submitting your information to the following link:

https://home.eease.adp.com/recruit/?id=1451671